(833) 317-6278
Book a Demo
Gabriela Gonzalez
Gray shield icon
Is this you?
Claim your profile.

Gabriela Gonzalez

AS SEEN ON
KGBT-TV (Harlingen, TX), NewsNation

Preston's Summary

Gabriela Gonzalez is a journalist based in Harlingen, Texas. She writes for KGBT-TV and her work has also been featured in KLST News, KAMC 28 News, KIAH-TV, KXAN, and Newschannel 9-KTSM. Gabriela covers a wide range of local news topics, including community events, infrastructure projects, crime reports, and human interest stories in the Harlingen area.

Preston is the artificial intelligence that powers the Intelligent Relations PR platform. Meet Preston

Geo Focus

Edinburg, United States (Local)

Coverage Attributes:

Beta
Breaking News: 22 %
Evolving Stories: 20 %
Event Coverage: 15 %
Government Announcement: 13 %
Press Release: 10 %

Themes Covered:

Not enough data icon

Not enough data

Most Recent Topics:

  • Event Management Tools
  • Event Planning
  • Food Safety
  • Legal Reform
  • Juvenile Detention
  • Construction Industry

Pitching Insights

Gabriela's coverage mainly focuses on local news, particularly crime and community events in the Texas area. She is likely to be interested in pitches that involve breaking news, evolving stories, government announcements, or press releases related to local crime, community events, infrastructure developments, and educational initiatives.

Those pitching should consider offering access to firsthand accounts of newsworthy incidents or providing expert analysis on relevant topics such as crime trends or infrastructure projects within the Texas region. Additionally, sources who can offer insights into family-related issues within the community may also be appealing given her interest in family and relationships.

This information evolves through artificial intelligence and human feedback. Improve this profile .

Journalists With Similar Coverage:

Based on similarity of content.
Tim Sullivan

Tim Sullivan

Sportswriter
*******an@***************.com
Publications
710 KURV, Page 17, Inc., The Star newspaper (Sun Prairie WI), PHILLY SPORTS NETWORK, LEO Weekly LLC
Most recent topics
Not enough data
Gloria Walker

Gloria Walker

News Reporter
******r@**********com
Publications
KRGV-TV
Most recent topics
Not enough data
Diana Eva Maldonado

Diana Eva Maldonado

Assistant News Director
********do@********tv
Publications
KGBT-TV (Harlingen, TX)
Most recent topics
Not enough data
Jesse Mendez

Jesse Mendez

******z@******om
Publications
KGBT-TV (Harlingen, TX), KXAN
Most recent topics
Not enough data
Sandy Rojas

Sandy Rojas

Publications
FOX West Texas, The Business Herald
Most recent topics
Not enough data
Brenda Villa

Brenda Villa

Publications
KRGV-TV
Most recent topics
Not enough data

Articles

DM2I

Omp25‐dependent engagement of SLAMF1 by Brucella abortus in dendritic cells limits acute inflammation and favours bacterial persistence in vivo

By: Gabriela Gonzalez, Pierre Gorvel, Stéphane Méresse, Clara Degos, Aurélie Gagnaire, Karina Alejandra Pasquevich, Juliana Cassataro, Sylvie Mémet, Alexia Papadopoulos, Vilma Arce

The strategies by which intracellular pathogenic bacteria manipulate innate immunity to establish chronicity are poorly understood. Here, we show that Brucella abortus outer membrane protein Omp25 specifically binds the immune cell receptor SLAMF1 in vitro. The Omp25-dependent engagement of SLAMF1 by B. abortus limits NF-κB translocation in dendritic cells (DCs) with no impact on Brucella intracellular trafficking and replication. This in turn decreases pro-inflammatory cytokine secretion and impairs DC activation. The Omp25-SLAMF1 axis also dampens the immune response without affecting bacterial replication in vivo during the acute phase of Brucella infection in a mouse model. In contrast, at the chronic stage of infection, the Omp25/SLAMF1 engagement is essential for Brucella persistence. Interaction of a specific bacterial protein with an immune cell receptor expressed on the DC surface at the acute stage of infection is thus a powerful mechanism to support microbe settling in its replicative niche and progression to chronicity.

© 2024 Intelligent Relations