Iman Mohamed is a Digital Producer at YES! Media. She focuses on topics related to environmental justice and sustainability, often exploring the intersection of technology and social issues. Iman has been featured in publications such as YES! Media, Paperity, the Fire Technology journal by Springer Nature, and PLOS.
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Iman's coverage mainly consists of profile features and expert commentary in the content & publishing and culture & society themes, with a focus on journalism, solutions journalism, YES! Magazine, and journalists. If reaching out to Iman Mohamed for potential coverage or collaboration opportunities, consider offering profile subjects who can provide unique insights into contemporary societal issues or experts who can offer valuable commentary within these thematic areas.
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By: Ain Shams, Iman Mohamed, Rasha Ahmed
Abstract Purpose The loss of flower quality after harvesting is a major concern in the floriculture industry.
By: David Powell, Neville Q. Verlander, Alice M. Nisbet, Hilary Kirkbride, Marie A. Chattaway, Iman Mohamed, Gemma C. Langridge, Andrew Nelson, Emma V. Waters
Abstract Modern knowledge regarding ‘healthy bacterial carriers' traces back to the legacy of Typhoid Mary, who unknowingly infected >50 people with enteric fever.
By: Sarah Aly, Talaat, Lerine Bahy-Eldin, Abdullah Ahmed Amin, Eladawy, Mohammed Saad Eldin, Al Sayed Abdel Salam, Sahar Samir Abdel, Faramawy, Iman Mohamed, Elshazly, Amel Abdel-Magied El, Mohammed
Background Portal vein thrombosis (PVT) is an important cause of portal hypertension in the pediatric age group with high morbidity rates due to its main complication—the upper gastrointestinal varices and hypersplenism. No identifiable cause can be found in more than half of the cases of extrahepatic portal vein obstruction (EHPVO). Thrombophilia is incriminated in 35% of cases of EHPVO in children. Objective This study aimed to determine the prevalence of hereditary thrombophilia in Egyptian children with PVT. Methods This cross-sectional study included all children with PVT attending Ain Shams University Children’s Hospital Hepatology Unit over 1 year. All patients had hereditary thrombophilia done including (protein C, protein S, antithrombin III activities; MTHFR (C677T), factor V Leiden, and prothrombin gene, G20210A mutations). In addition to assess the clinical impact of hereditary thrombophilia on the pattern and severity of PVT, the cohort was categorized according to thrombophilia screening into two groups: patients with hereditary thrombophilia (44/70) and those without hereditary thrombophilia (26/70). This categorization enabled comparative analysis of radiological and endoscopic findings between the two groups. Results Our cohort included 70 children with PVT (median age 3 years; the male-to-female ratio among the studied children was 1.9:1, with males representing 65.7%. A positive family history of venous thrombosis was identified in one patient (1.4%). Hematemesis was the most frequent presenting symptom (64.3%), followed by abdominal enlargement (35.7%). Thrombophilia disorders were detected in 62.9% of cases, predominantly protein C deficiency (42.9%) and homozygous MTHFR mutations (22.9%). Patients with thrombophilia showed significantly greater portal vein attenuation with cavernous transformation (84.4% versus 65.4%, p = 0.039). Conclusion Hereditary thrombophilia may be a contributing factor of PVT. Therefore, hereditary thrombophilia screening is recommended in this population.
